"The preliminary results suggest that SARS-CoV-2 replicates in bacterial growth"
https://t.co/7pmlPCo8B2
Italian researchers: vaccines will not work against SARSCoV2 because this virus does not only replicate in human cells like other viruses, this one replicates through bacteria too. This is the fundamental reason why antibiotics work & vaccines will not:
"The preliminary results suggest that SARS-CoV-2 replicates in bacterial growth"
https://t.co/7pmlPCo8B2
More from Robin Monotti FRSA ⭐
I’m not just a lockdown sceptic. I know lockdowns increase infections rather than decrease them because I read research first hand. I’m a citizen in a democracy who wants Government policy to be evidence-based, not epidemiological modelling based. Crazy, I know.
Lockdowns increase infections because they lower immunity & therefore increase the disease. After hospitals & care homes households account for the largest number of transmissions. Schools/universities act as a break in transmission of the disease. All evidence based.
Transmission does not mean infection. I can transmit SARSCoV2 but I can't transmit Covid19. Whether SARSCoV2 develops at all into mild or severe Covid19 depends entirely on the immune system of the recipient. Early treatment prevents severe Covid19, the right treatment cures it.
"Children act more as a brake on infection," said Prof. Reinhard Berner, the head of pediatric medicine at Dresden University Hospital and leader of the study. "Not every infection that reaches them is passed
Closing schools increases transmission of respiratory viral infection because children act as a break on the community transmission of the virus. We have known this since 1918:
https://t.co/TPRYQ1LAAJ
Lockdowns increase infections because they lower immunity & therefore increase the disease. After hospitals & care homes households account for the largest number of transmissions. Schools/universities act as a break in transmission of the disease. All evidence based.
Transmission does not mean infection. I can transmit SARSCoV2 but I can't transmit Covid19. Whether SARSCoV2 develops at all into mild or severe Covid19 depends entirely on the immune system of the recipient. Early treatment prevents severe Covid19, the right treatment cures it.
"Children act more as a brake on infection," said Prof. Reinhard Berner, the head of pediatric medicine at Dresden University Hospital and leader of the study. "Not every infection that reaches them is passed
Closing schools increases transmission of respiratory viral infection because children act as a break on the community transmission of the virus. We have known this since 1918:
https://t.co/TPRYQ1LAAJ
I have now re-examined this document:
It clearly does indicate both the risks of bacterial infection & to prescribe broad spectrum antibiotics as part of treatment:
"Collect blood cultures for bacteria that cause pneumonia and sepsis, ideally before antimicrobial therapy. DO NOT
delay antimicrobial therapy"
"6. Management of severe COVID-19: treatment of co-infections
Give empiric antimicrobials [broad spectrum antibiotics] to treat all likely pathogens causing SARI and sepsis as soon as possible, within 1 hour
of initial assessment for patients with sepsis."
"Empiric antibiotic treatment should be based on the clinical diagnosis (community-acquired
pneumonia, health care-associated pneumonia [if infection was acquired in health care setting] or sepsis), local epidemiology &
susceptibility data, and national treatment guidelines"
"When there is ongoing local circulation of seasonal influenza, empiric therapy with a neuraminidase inhibitor [anti-viral influenza drugs] should
be considered for the treatment for patients with influenza or at risk for severe disease."
On the 19th March 2020 the WHO released this guidance intended for healthcare workers (HCWs), healthcare managers and IPC teams at the facility level & at national and district/provincial level:https://t.co/C4aV2BnMPj pic.twitter.com/tCk1EyLskV
— Robin Monotti (@robinmonotti) December 21, 2020
It clearly does indicate both the risks of bacterial infection & to prescribe broad spectrum antibiotics as part of treatment:
"Collect blood cultures for bacteria that cause pneumonia and sepsis, ideally before antimicrobial therapy. DO NOT
delay antimicrobial therapy"
"6. Management of severe COVID-19: treatment of co-infections
Give empiric antimicrobials [broad spectrum antibiotics] to treat all likely pathogens causing SARI and sepsis as soon as possible, within 1 hour
of initial assessment for patients with sepsis."
"Empiric antibiotic treatment should be based on the clinical diagnosis (community-acquired
pneumonia, health care-associated pneumonia [if infection was acquired in health care setting] or sepsis), local epidemiology &
susceptibility data, and national treatment guidelines"
"When there is ongoing local circulation of seasonal influenza, empiric therapy with a neuraminidase inhibitor [anti-viral influenza drugs] should
be considered for the treatment for patients with influenza or at risk for severe disease."
More from Category c19
Let's talk about MASKS!
Thread 1:
Masks increase mortality because breathing through them nebulizes aerosols into smaller ones which bypass mucosal immunity & reach all the way into the alveoli, leading to acute respiratory distress syndrome (ARDS).
"Aerosols..within the most breathable size range between 0.5 & 5 μm, can carry SARS-CoV-2 deep to the terminal alveoli..if this transmission pathway does exist, it would bypass the mucociliary clearance & incubation period of the virus in the upper
The filtration material itself of N95's average pore size ~0.3−0.5 μm does not block finer aerosol laden with virions penetration, not to mention surgical masks.
Thread 1:
If you study the field you will see viral nebulization is a technique used to get higher infectivity of viruses. This is used with ventilator patients suffering from pseudomonas aeruginosa infections. Nebulize bacteriaphage to get deep into the lungs and kill bacteria. pic.twitter.com/82lNRMrXl4
— Kevin McKernan \U0001f642 (@Kevin_McKernan) October 24, 2020
Masks increase mortality because breathing through them nebulizes aerosols into smaller ones which bypass mucosal immunity & reach all the way into the alveoli, leading to acute respiratory distress syndrome (ARDS).
"Aerosols..within the most breathable size range between 0.5 & 5 μm, can carry SARS-CoV-2 deep to the terminal alveoli..if this transmission pathway does exist, it would bypass the mucociliary clearance & incubation period of the virus in the upper
The filtration material itself of N95's average pore size ~0.3−0.5 μm does not block finer aerosol laden with virions penetration, not to mention surgical masks.
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So it's now October 10, 2018 and....Rod Rosenstein is STILL not fired.
He's STILL in charge of the Mueller investigation.
He's STILL refusing to hand over the McCabe memos.
He's STILL holding up the declassification of the #SpyGate documents & their release to the public.
I love a good cover story.......
The guy had a face-to-face with El Grande Trumpo himself on Air Force One just 2 days ago. Inside just about the most secure SCIF in the world.
And Trump came out of AF1 and gave ol' Rod a big thumbs up!
And so we're right back to 'that dirty rat Rosenstein!' 2 days later.
At this point it's clear some members of Congress are either in on this and helping the cover story or they haven't got a clue and are out in the cold.
Note the conflicting stories about 'Rosenstein cancelled meeting with Congress on Oct 11!"
First, rumors surfaced of a scheduled meeting on Oct. 11 between Rosenstein & members of Congress, and Rosenstein just cancelled it.
He's STILL in charge of the Mueller investigation.
He's STILL refusing to hand over the McCabe memos.
He's STILL holding up the declassification of the #SpyGate documents & their release to the public.
I love a good cover story.......
The guy had a face-to-face with El Grande Trumpo himself on Air Force One just 2 days ago. Inside just about the most secure SCIF in the world.
And Trump came out of AF1 and gave ol' Rod a big thumbs up!
And so we're right back to 'that dirty rat Rosenstein!' 2 days later.
At this point it's clear some members of Congress are either in on this and helping the cover story or they haven't got a clue and are out in the cold.
Note the conflicting stories about 'Rosenstein cancelled meeting with Congress on Oct 11!"
First, rumors surfaced of a scheduled meeting on Oct. 11 between Rosenstein & members of Congress, and Rosenstein just cancelled it.
Rep. Andy Biggs and Rep. Matt Gaetz say DAG Rod Rosenstein cancelled an Oct. 11 appearance before the judiciary and oversight committees. They are now calling for a subpoena. pic.twitter.com/TknVHKjXtd
— Ivan Pentchoukov \U0001f1fa\U0001f1f8 (@IvanPentchoukov) October 10, 2018
The entire discussion around Facebook’s disclosures of what happened in 2016 is very frustrating. No exec stopped any investigations, but there were a lot of heated discussions about what to publish and when.
In the spring and summer of 2016, as reported by the Times, activity we traced to GRU was reported to the FBI. This was the standard model of interaction companies used for nation-state attacks against likely US targeted.
In the Spring of 2017, after a deep dive into the Fake News phenomena, the security team wanted to publish an update that covered what we had learned. At this point, we didn’t have any advertising content or the big IRA cluster, but we did know about the GRU model.
This report when through dozens of edits as different equities were represented. I did not have any meetings with Sheryl on the paper, but I can’t speak to whether she was in the loop with my higher-ups.
In the end, the difficult question of attribution was settled by us pointing to the DNI report instead of saying Russia or GRU directly. In my pre-briefs with members of Congress, I made it clear that we believed this action was GRU.
The story doesn\u2019t say you were told not to... it says you did so without approval and they tried to obfuscate what you found. Is that true?
— Sarah Frier (@sarahfrier) November 15, 2018
In the spring and summer of 2016, as reported by the Times, activity we traced to GRU was reported to the FBI. This was the standard model of interaction companies used for nation-state attacks against likely US targeted.
In the Spring of 2017, after a deep dive into the Fake News phenomena, the security team wanted to publish an update that covered what we had learned. At this point, we didn’t have any advertising content or the big IRA cluster, but we did know about the GRU model.
This report when through dozens of edits as different equities were represented. I did not have any meetings with Sheryl on the paper, but I can’t speak to whether she was in the loop with my higher-ups.
In the end, the difficult question of attribution was settled by us pointing to the DNI report instead of saying Russia or GRU directly. In my pre-briefs with members of Congress, I made it clear that we believed this action was GRU.
